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As a result of primary infection, the body produces antibodies to the particular type of HSV involved, preventing a subsequent infection of that type at a different site.

In HSV-1-infected individuals, seroconversion after an oral infection prevents additional HSV-1 infections such as whitlow, genital herpes, and herpes of the eye.

__________________________________________________ Note: Not all genital herpes statistics are consistent from study to study.

Most say 25% of American women have the virus, and 20% of American males. The studies with slightly lower numbers refer to people ¹ (Source: ² (Source: ³ (Source: ⁴ (Source: ⁵ (Source: ⁶ (Source:

Please also keep in mind that these statistics account for genital herpes caused by the HSV-2 strain. Unfortunately, when clinicians give “standard” STD check-ups, they don’t include tests for HSV-1 or HSV-2, unless it is specifically requested.

This only exacerbates herpes’ prevalence and the ignorance around it. Many couples have a healthy sex life and still never transmit it to the uninfected partner.

It is often associated with impaired immune function (e.g.

This theory has been contested, however, since HSV is detected in large numbers of individuals having never experienced facial paralysis, and higher levels of antibodies for HSV are not found in HSV-infected individuals with Bell's palsy compared to those without.

In the presence of a certain gene variation (APOE-epsilon4 allele carriers), HSV-1 appears to be particularly damaging to the nervous system and increases one's risk of developing Alzheimer's disease.

In all cases, HSV is never removed from the body by the immune system.

Following a primary infection, the virus enters the nerves at the site of primary infection, migrates to the cell body of the neuron, and becomes latent in the ganglion.

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